TLO 2.4 Gastrointestinal/Bowel Child Flashcards

1
Q

Functional differences in the colon of a child

A

Large intestine of children is shorter than adult
Less epithelial lining to absorb water from feces resulting in softer stool and more rapid peristaltic waves
Fever increases the rate of peristalsis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Stool frequency in infants, bottle vs breast, color

A

Bottle: 1-3 per day
Breast: 4-6 per day
Color: dark green first week then yellow

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Causes of constipation in children

A
Changes in diet
Dehydration
Lack of exercise
Emotional stress
Medication
Pain from anal fissure
Excessive dairy intake
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Diagnosis of constipation

A

Abdominal x-ray: enlarge rectum with stool and gas present
Rectal exam: rarely performed r/t emotional impact
Thorough history usually sufficient to diagnosis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Constipation treatment

A

Best through prevention

Dietary modifications:
increase water and fiber intake
decrease sugar and milk intake

Teach:
sit child on toilet 5-10 min approx. 30 min after breakfast and dinner
offer charts and prized to reward success
proper use of enema is physician suggest

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Infectious Gastroenteritis (infectious diarrhea): causes

A

Rotavirus: most common viral cause
Giardia: most common pathogen in daycare setting

Communicable diarrhea
Massive fluid and electrolyte loss
Sepsis
Death

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Giardia: incubation, spread, s/s, diagnosis, treatment

A

Incubation: 1-2 weeks. Most common parasitic diarrhea

Spread in contaminated food and water

S/S: afebrile, abdominal distention, cramps and gas

Diagnosis: ova and parasites found in stool

Treatment: Flagyl x7 days

Contact precautions

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Rotavirus: incubation, spread, s/s, diagnosis, treatment

A

Incubation: 1-3 days. Common in winter months

Spread by fecally contaminated food

S/S: vomiting, diarrhea, low grade fever. Last 3-7 days

Diagnosis: virus in stoll detected enzyme immunoassay

Treatment: no pharmacologic treatment, supportive care, maintain hydration, rotavirus vaccine at 2, 4, 6 months

Contact precautions

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Gastrointestinal/bowl assessment finding?

A
Assess hydration status
Dry mucus membranes
Poor skin turgor, crying without tears
Sunken fontanel in infants
Increased respiratory rate r/t metabolic acidosis
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Gastroenteritis teaching

A

Wash hands frequently
Child to use separate bathroom if available
Administer PO fluids to rehydrate
Avoid fruit juices, cola, tea, sugary drinks, sports drinks
Continue breast mild and formula
No OTC without asking doctor

Call PCP when:
diarrhea worsens/blood
s/s dehydration
vomiting increases and cannot keep down any fluids
child reports severe abdominal pain
child hasn't urinated for over 6 hours
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Intussusception, what is it?

A

Folding of one part of the intestine into another causing bowel obstruction. Occurs around 3-6 months
Relatively rare usually but when occurs it is and emergency
Recurrence does sometimes occur

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Causes of intussusception?

A

Unknown
Contributing factors:
Preexisting URI or other vial infection
Pathologic condition within the colon (mass/defect)
More common in boys than girls
Children with cystic fibrosis are more susceptible

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Intussusception diagnosis and prognosis?

A

X-ray: gas patterns like bowel obstructions
Ultrasound: identifies location of intussusception
Barium or air enema: reduces the obstruction (both diagnostic and treatment) 80-95% effective

Prognosis: death is uncommon, 80% success with nonsurgical

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Intussusception assessment

A

History reveals sudden crying and flexing legs in infants
Pain that come and goes and progresses to constant severe
Bloody mucus currant jelly stools/diarrhea, vomiting
Sausage shaped abdominal mass

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Intussusception assessment >12-24 hours

A
Shock
Sepsis
Listlessness
Fever
Decrease LOC
Increased heart rate
Blood pressure changes
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Intussusception treatment

A

Spontaneous reduction occurs in <10% of cases

Conservative treatment:
-Radiologist guided pneumo enema (air enema)
-Barium enema, 80-95% effective
Ultrasound guided hydrostatic (saline) enema
-Prior to hydrostatic procedure: IV fluids, NG decompression, antibiotic therapy

Surgery is conservative treatment fails

17
Q

Intussusception nursing interventions

A

Observe passage of barium following BE
Monitor stools, passage of normal stool color indicates resolution
Assess for s/s bowel obstruction
Monitor for normal bowel function
NPO post op until bowel function returns
Listen to parents description of symptoms

18
Q

Hirschsprung disease, what is it?

A

AKA Congenital Aganglionic Megacolon
Strong heredity component, Down Syndrome
Results from absence of ganglion nerve cells in the rectum that regulate the activity of the colon
Can be acute, life threatening or chronic condition
Major cause of lower bowel obstruction in infants

19
Q

Hirschsprung disease assessment

A
Pellet or ribbon like foul smelling stools
Distended abdomen
Refusal to feed/intolerance
Bilious vomiting
Delay in passage of meconium
Growth failure
Constipation problems since birth
Episodes of diarrhea/vomiting
20
Q

Hirschsprung disease, signs of enterocolitis

A

Inflammation of the small bowel and colon
Fever
Abdominal distention
Diarrhea
May be severe with life threatening dehydration or sepsis

21
Q

Hirschsprung disease diagnostic tests

A

Rectal exam: tight anal sphincter and absence of stool followed by explosive release of gas and stool

Barium enema: reveals change in size in color

Anorectal manometric exam: tube inserted rectally has balloon that filled with air to measure function of muscles and nerves inside rectum

Definitive diagnosis: punch biopsy shows absence of ganglionic cells

22
Q

Hirschsprung disease medical management

A

Relieving chronic constipation

  • stool softeners
  • isotonic enema
  • low residue diet

Surgical intervention

  • removes aganglionic portion of bowel
  • 2 stage surgery most cases

Botox: to relax anal sphincter

23
Q

Hirschsprung disease nursing intervention

A
No rectal temp
Assess change in abdominal circumference
Bowel sounds
Prepare child for surgery
Prevent infection
Maintain skin integrity
Maintain nutrition and hydration
Reduce pain
NPO, NG tube, intermittent suction
24
Q

Appendicitis, what is it? Causes?

A

Inflammation and infection of the vermiform appendix
Causes:
lymphoid swelling r/t viral infection, impacted fecal material, foreign bodies, parasites
often no cause found

25
Q

Appendicitis assessment

A

Cardinal symptom: pain increasing in intensity RLQ at McBurney point

Associated s/s:
N/V
Anorexia
Diarrhea/constipation
Fever/chills
If perforation occurs report of pain relief initially followed by increased RLQ pain
26
Q

Appendicitis diagnosis/treatment

A

Diagnosis:
Based on c/o classic pain at McBurney’s point
WBC count of 15,000-20,000 supports clinical findings
Ultrasound or CT shows enlarged appendix

Treatment:
Appendectomy

27
Q

Appendicitis nursing intervention

A

Uncomplicated appendicitis:
V/S
Cold application, NOT heat (can potentiate rupture)
IV fluids preop and NPO

Ruptured appendix:
NG tube to decompress preop and drain gastric contents post op
IV antibiotics
Monitor drains if present
Pain management
Monitor WBC count