Week 4 - Cardiogenic Shock Flashcards

1
Q

what is cardiogenic shock?

A
  • occurs when the hearts pumping ability is compromised to the point at which it cannot maintain cardiac output and adequate tissue perfusion
  • HEARTS ABILITY TO CONTRACT AND TO PUMP BLOOD IS IMPAIRED LEADING TO DECREASED CO
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2
Q

COMMON CAUSES FOR

CARDIOGENIC SHOCK

A
  • Myocardial Infarction (most common)
  • Arrhythmias
  • Cardiomyopathies
  • Severe Hypoxemia
  • Acidosis
  • Cardiac Surgery
  • Valvular Issues

HINT: ‘MACSAC V’

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3
Q

_____________ is the most common

cause of cardiogenic shock

A

Myocardial Infarction

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4
Q

OVERALL GOALS R/T MEDICAL

MANAGEMENT OF PT

W/CARDIOGENIC SHOCK

A
  • Limit further damage to the myocardium and preserve healthy tissue
  • Improve cardiac output
  • Identify underlying cause and correct it
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5
Q

Name three (3) types of Colloids

A

Albumin (Naturally Occurring) Dextran , Hetastarch (synthetics)

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6
Q

How does albumin help with shock?

A
  • Albumin is a large molecule
  • Water is attracted to the large molecule

If we give a patient Albumin then we are able to HOLD onto fluid in the vascular space.

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7
Q

Albumin is a naturally occurring colloid.

What are the two SYNTHETIC colloids?

A
  • Dextran
  • Hetastarch
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8
Q

Do you need two nurses to administer Albumin?

A

No. Even though ALBUMIN is a “blood product” it does not go through all the same checks as other blood products do.

  • you do NOT need two nurses for administration, and
  • you do NOT need any special tubing or filter tubing for administration.

IMPORTANT TO NOTE: Patients that often refuse “blood products” (ie, Jehovahs Witness) WILL refuse Albumin because the albumin is synthesized out from blood that is donated

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9
Q

Colloids can affect platelet adhesion causing//

A

causing our patients an increased risk of bleeding

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10
Q

(True / False)

Colloids <strong>(Albumin, dextran, Hetastarch)</strong> reduce platelet

adhesiveness and is associated with reductions is blood coagulation.

A

TRUE

  • We should be monitoring the clients:(values listed below are the normal lab values)
    • PT 10-15 seconds
    • INR 1 - 1.2 seconds
    • Platelets 150,000 - 400,000 /mm3
    • aPTT < 35 sec
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11
Q

True / False Primary medications for cardiogenic shock consist mostly of DILATORS and INOTROPES?

A

TRUE

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12
Q

In cardiogenic shock–should we stimulate the BETA receptors?

A

NO–because this will end up making the heart work harder than it needs to

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13
Q

Why are DILATORS good for pts with Cardiogenic shock–

A

It dilates leaving less work load on the heart

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14
Q

if cardiogenic shock patient is BRADYCARDIC we might need _________ and/or a __________

A

ATROPINE PACEMAKER

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15
Q

>

Medical Management of

Cardiogenic Shock

A
  • Supplement the hearts own functioning w/ meds/devices
  • Control Chest Pain
  • Fluid Management
    • Be conservative (dont want to overload their heart)
  • Diuretics
    • may be needed to get rid of excess volume so heart can function better
  • Colloids
    • Albumin <strong>(naturally occurring colloid)</strong>
    • Dextran (synthetic)
    • Hetastarch (synthetic)
  • VASOACTIVE MEDS
    • Primarly given:
      • DILATORS
      • INOTROPES (Dobutamine—strictly there to strengthen the contraction but doesnt increase heart rate)
    • (in extreme cases vasoconstriction is necessary to maintain MAP)
  • CONTROLLING/SUPPORTING HEART RATE/RHYTHM
    • ​If Bardycardic- use ATROPINE and possibly a pacemaker
  • ASSISTIVE DEVICES
    • IABP
    • LVAD
    • ECMO
  • LABS
    • Brain Natriuretic Peptide (BNP)
      • >100 pg/mL(can be indicative of heart failure)
    • Creatinine Kinase (CK)
      • > 200 U/L (indicative of muscle damage)
    • Troponin
      • > 0.4 ng/mL
  • EKG
  • ECHO (u/s of heart)
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16
Q

Important Labs / Diagnostics to check (Cardiogenic Shock)

A

NORMAL BNP <100 pg/mL **Labs will be ELEVATED NORMAL CK 22 - 198 U/L **Labs will be ELEVATED NORMAL TROPONIN 0 - 0.4 ng/mL **Labs will be ELEVATED **ECHOCARDIOGRAM **EKG

17
Q

medications that act directly on the smooth muscle, relaxing arterioles and veins

A

vasodilators

18
Q

How to Vasodilators affect the heart

A
  • acts directly on the smooth muscle, relaxing both arterioles and veins
    • RESULTS In…..decreased O2 demands of the heart
      • Decreased PRELOAD
      • Decreased AFTERLOAD
    • <strong>Examples: </strong> Nitroglycerin, Nitroprusside
19
Q

Nursing management of Cardiogenic Shock

A
  • Recognition of S/S
    • Poor perfusion? Cyanosis? Chest pains? Arrhythmias?
  • Monitoring hemodynamic status
    • CVP- make sure pt isnt overloaded with fluid
    • Pulmonary Artery Catheter
    • Vasodilators can lead to rebound tachycardia and increases O2 demands of the heart
  • Administering fluids and medications
    • Closely watch for fluid overload
    • Complications of vasoactives
  • Management of Assistive Devices
20
Q

(TRUE / FALSE) Pulmonary Artery Catheter sits in the PA and measures the pressures in the lungs and left side of the heart and measures cardiac output

A

TRUE

21
Q

Reasons for a Pulmonary Artery Catheter

A

1) Early detection/treatment of life threatening conditions of the heart and lung
2) Evaluation of clients immediate response to treatment
3) Evaluation of the effectiveness of Central Venous function

22
Q

Assistive devices (3) sometimes

used in the management of

Cardiogenic Shock

A

Intra-Aortic Balloon Pump (IABP)

Left Ventricular Assist Device

Extracorporeal membrane oxygenation

23
Q

What does the IABP do?

Intra-Aortic Balloon Pump

A
  • Mechanical device used to:
    • Increase myocardial oxygen perfusion
    • Increase cardiac output

Placed through femoral artery via sheath

filled with helium

sits distal to left subclavian artery

<em><u>INFLATES on diastole to IMPROVE LV Volume and perfuse coronary arteries</u></em>

DEFLATES on systole to pull blood out of LV (via vacuum effect)–decreased afterload

24
Q

Complications of IABP

A
  • Lower Extremity Compromise
    • Ischemia
    • Compartment Syndrome-Pain, Pallor, Pulse, Paresthesia, Paralysis
    • Infection
    • Aortic Dissection (tear in Aorta)
    • Device Malfunction
25
Q

Indications for IABP

A
  • Cardiogenic Shock- b/c it supports functioning of the heart
  • Post-Op cardiac surgery- as temporary/supportive measure
  • Cardiac<em> (mechanical)</em> defects
    • Mitral Valve Regurgitation
    • septal perforation
26
Q

Circulatory DEVICE that is used

to partially (or completely) replace

the function of a failing heart

A

Left Ventricular Assist Device (LVAD)

27
Q

What is a LVAD

A

Left Ventricular Assist Device (LVAD)

–it is used to partially or completely replace the function of a failing heart

28
Q

How long can you use a LVAD?

A
  • SHORT TERM:
    • following a MI or heart surgery to prevent / treat cardiogenic shock
  • LONG TERM:
    • used with severe Heart Failure (EF 5 - 10%)
    • often used as a bridge to transplant
29
Q

What is ECMO

A
  • Extracorporeal Membrane Oxygenation –advanced life support technique –provides long term breathing / heart support *blood is taken outside the body-oxygenate, warmed, CO2 removed, then pumped back into the body
30
Q

Types (2) of set-up for the ECMO

A

VENO-ARTERIAL (vein to artery) —Primarily cardiac failure w/ respiratory support VENO-VENOUS (Vein to Vein) —Primarily for Respiratory failure (ARDS)

31
Q

CARIOGENIC SHOCK (RIGHT SIDED)

<em><strong>FLUIDS</strong>…?? </em>

<em><strong>First-Line Agent(s):… Second-Line Agent(s)…??</strong></em>

A

1) FLUID:

Crystalloid (20 mL / kg)

2) 1st line tx:

Vasopressors directed at cause of right heart failure

Dopamine (with signs of shock)-<em>increased rate and contractility</em>

Dobutamine (w/o signs of shock)-<em><strong>increased contractility</strong></em>

3) 2nd line tx:

additional Vasopressors PRN

Dopamine -<em>increased rate and contractility</em>

Epinephrine<em><strong>-increased rate and contractility</strong></em>

Milrinone (severe CHF or RV infarct)

32
Q

CARIOGENIC SHOCK (LEFT - SIDED) FLUIDS…?? First-Line Agent(s):… Second-Line Agent(s)…??

A

1) FLUID:

IV fluids as appropriate-use caution with CHF

2) 1st line tx:

Dobutamine (w/o signs of shock)-increased contractilit

Norepinephrine (with signs of shock)- initiates vasoconstriction

3) 2nd line tx:

Dobutamine and Norepinephrine

Epinephrine-<strong>increased rate and contractility</strong>

Milrinone <em><strong>(severe CHF or RV infarct)</strong></em>

33
Q

LVAD

Left Ventricular

Assist Device

A
34
Q

HEALTH PROMOTION / DISEASE PREVENTION

“what education would you provide to assist the patient on

how to reduce the risk of cardiogenic shock?”

A
  • Educate client on how they can reduce their RISK for MYOCARDIAL INFARCTION (MI)
    • Exercise
    • diet
    • stress reduction
    • smoking cessation
35
Q
A