Unit 3 - 3.8-3.11 Flashcards

1
Q

Characterized by infiltration of mononuclear cells, usually accompanied by fibroplasia, and occurs when the acute inflammatory reaction does not resolve the original injury:

A

Chronic inflammation

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2
Q

What are the purposes of fibroplasia?

A
  • walls off

- inflammatory cells (creation of small vessels)

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3
Q

What is the typical fate of fibroplasia?

A

collagen content increase, fibrous tissues matures and contracts

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4
Q

If there is significant fibrosis, the surface of the chronic inflammation will be:

A

pitted

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5
Q

Why can chronic inflammation be nodular?

A

presence of abscesses or granulomas

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6
Q

What’s the general colorization of chronic inflammation?

A

grey to white and firm

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7
Q

What does the type of chronic inflammation depend on me?

A
  • location in body

- infected vs aseptic

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8
Q

Collection of pus in a newly formed cavity usually due to bacteria

A

abscess

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9
Q

What types of cells help form a wall around the neutrophilic infiltrate after 2-3 days? What is this structure called?

A

fibroblasts, angioblast

- pyogenic membrane

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10
Q

Why does the inner surface of an abscess produce pus?

A

provides fresh neutrophils

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11
Q

What is the function of the outer surface of a pyogenic membrane?

A

barrier to prevent spread

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12
Q

What are the 3 basic fates of an abscess?

A
  • scar
  • expands
  • empties
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13
Q

Why can an abscess expand?

A

because bacteria fight back

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14
Q

How does an abscess empty?

A

into a fistula to another body cavity or to a surface

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15
Q

How can abscess turn into a scar?

A

bacteria killed, pus resorbed, pyogenic membrane shrinks

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16
Q

spreading of inflammation into the subcutis

A

cellulitis

17
Q

spreading of inflammation along parallel tissue planes

A

phlegmon

18
Q

the accumulation of pus in a pre-existing cavity such as the pleural cavity

A

empyema

19
Q

What type of cell predominates in granulomatous inflammation?

A

macrophages

20
Q

A response to indigestible, poorly degradable or persistent antigenic agents

A

granulomatous inflammation

21
Q

How do multinucleated giant cells form?

A

fusion of macrophages

22
Q

Diffuse infiltrate of macrophages and.or multinucleated giant cells and accompanied by other inflammatory cells, especially lymphocytes:

A

granulomatous inflammation

23
Q

Large macrophages that resemble epithelial cells and are located in the center of granulomas. Function is only to secrete mediators:

A

epitheloid macrophages

24
Q

List the three common subtypes of granulomatous inflammation:

A
  • pyogranulomatous inflammation
  • eosinophilic granulomatous inflammation
  • caseating granulomas
25
Q

Neutrophils seen in the center of granulomas

A

pyogranulomatous inflammation

26
Q

Eosinophils seen in the center of granulomas:

A

eosinophilic granulomatous inflammation

27
Q

granulomas with large central areas of necrosis; suggestive of TB:

A

caseating granulomas

28
Q

proliferation of fibrous CT (fibroblasts and angioblasts)

A

fibroplasia

29
Q

Chronic inflammation is typically characterized by a combination of:

A

mononuclear cells and fibroplasia (usually, but not always)

30
Q

Why can the surface of chronic inflammation appear pitted?

A

fibrous CT contracts over time

31
Q

What are some of the contributing factors to the firmness of tissue with chronic inflammation?

A
  • fibrous CT

- sheer number of inflammatory cells

32
Q

What is fibroplasia also commonly called?

A

granulation tissue

33
Q

Part of the healing process in tissues that can’t be completely restored to normal:

A

fibroplasia

34
Q

pathologic channel that erodes from an abscess or body cavity into another cavity or to the body surface

A

fistula

35
Q

Rounded mass-like aggregates of macrophages; in other words, granulomas are a nodular form of granulomatous inflammation:

A

nodular granulomas