THINGS SHE STRESSED PT 2-From Steve's Study Guide-Lecture 2/27 Flashcards Preview

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Flashcards in THINGS SHE STRESSED PT 2-From Steve's Study Guide-Lecture 2/27 Deck (34)
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1

3 manifestations of P waves in escape rhythms?

One lacks a P-Wave, one has inverted P-wave, and you have one where it appears after the QRS.

2

Atrial Dysrhythmias common cause of ______

stroke

3

Atrial Dysrhythmias-If in the right atria, and travels to lungs, they would get a______

pulmonary embolism

4

Atrial Dysrhythmias-Most often, the concern is the ____atrial clot, it can move from LA into the arterial circulation and get to the brain where an ______ occurs

LEFT; EMBOLIC STROKE

5

If a patient has Premature Atrial contraction it’s just he _____contraction that’s ectopic (does not come from SA node)

ATRIAL

6

comes from Purkinje fibers, happens when SA and AV node fail to fire, the Purkinje will fire to get the ventricles to move. It’s about 15 to 40 BPM

Ventricular Escape rhythm

7

A FIB and Flutter can BOTH cause ______ formation

thrombi

8

Try to suppress A-Fib with ______ and ________

beta blockers; anti arrythmics

9

There’s a
bizarre QRS
(it can have a negative deflection) it can
have a polymorphic-
one up, one down as at left

PVC's

10

a type of V-Tach/DEADLY

Torsades de Pointes

11

P waves are not
associated with the QRS complexes.

V TACH

12

no identifiable QRS complexes

V Fib

13

characterized by
progressive prolongation of the PR interval until
one P wave is not conducted;
associated with AV
nodal ischemia

Second Degree Type 1 Atrioventricular Block

14

Every QRS has a P wave but not every P wave has a QRS

Second Degree Type 2 Atrioventricular Block

15

can remain in the chest for 18 months or longer which
can monitor for arrhythmias.

DS loop recorder for 3rd degree block

16

PR interval will be greater than .2

First Degree Atrioventricular Block

17

How to treat coronary heart disease?

Baby aspirin, no more than 81mg, and a statin
-a cholesterol lowering drugs. Statins given
to prevent heart attack, slow down inflammatory process in these plaques, slow down the coagulation.

18

Characterized by stenotic atherosclerotic coronary vessels
.
Onset of anginal pain is generally
predictable and elicited by similar stimuli each time
.
Relieved by rest and nitroglycerin

Stable/typical angina

19

May progress to acute infarction

Unstable/crescendo angina

20

Onset of symptoms is unrelated to physical or emotional exertion, heart rate, or other
obvious causes of increased myocardial oxygen d
emand

Prinzmental/variant angina

21

Characterized by vasospasms and abnormal calcium flux

Prinzmental/variant angina

22

Acute coronary syndrome is an umbrella term for ____ and ______

unstable angina; MI

23

Any of the coronary heart syndromes may produce sudden ______

cardiac death

24

acute full occlusion of a coronary artery that leads to death of the myocardium tissue due to lack
of oxygen

STEMI

25

For candidates getting acute reperfusion therapy, how does the cardiologist do it?

angioplasty and stent

26

Patients preventing with symptoms of unstable angina with
no ST segment elevation, these patients have
______

NSTEMI

27

S/S
Severe crushing, excruciating chest pa
in that may radiate to the arm shoulder, jaw, or back
accompanied by nausea, vomiting, diaphoresis (sweating), shortness of breath

ACS

28

In women (and the elderly
and those with diabetic neuropathies)
Atypical symptoms, including fatigue

ACS

29

ECG changes for ACS include _____ elevation, large ____- waves, inverted _____ waves, and deep Q waves which equal old _____

ST; Q; T; MI

30

What's the treatment for ACS?

Decrease myocardial oxygen demand; beta blockers; PCI (angioplasty with a stent); if not PCI then CABG