Exam II - Eucoccidiorida Flashcards Preview

RUSVM Parasitology > Exam II - Eucoccidiorida > Flashcards

Flashcards in Exam II - Eucoccidiorida Deck (21)
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1
Q

Which parasitic group does Eimeria leukarti fall into?

A

Protozoan

2
Q

Describe the Eimeria leukarti oocyst

A

very large, 80 x 60 um, with a thick shell and distinct micropyle. The shell can come off

3
Q

What is/are the host(s) for Eimeria leukarti?

A

all equids

4
Q

What are the three phases in the Eimeria leukarti life cycle? What is the PPP for the 3 phases?

A
  1. Sporulation
  2. Asexual reproduction: infection & schizogony
  3. sexual reproduction: gametogony and oocyst formation

PPP is 15 days

5
Q

What is/are the site(s) of infection for Eimeria leukarti?

A

small intestine

6
Q

Describe any pathogenesis associated with Eimeria leukarti

A

inflammatory changes in mucosa, disruption of villous architecture

7
Q

Describe any clinical signs associated with Eimeria leukarti. Who is most susceptible?

A

Intermittent diarrhea. Foals are most susceptible.

8
Q

How would you diagnose Eimeria leukarti?

A

Hard to diagnose because eggs are heavy. Sugar flotation of sedimentation methods are used

9
Q

Treatment & prevention options for Eimeria leukarti?

A

Antiprotozoals and supportive therapy

10
Q

What is the common name of the disease caused by Sarcocystis neurona?

A

Equine protozoal myeloencephalitis (EPM)

11
Q

What is/are the host(s) for Sarcocystis neurona?

A

Definitive host is opossum

Intermediate hosts are cats, striped skunks, raccoons, etc…

Accidental (aberrant) host is horse

12
Q

The Sarcocystis neurona life cycle includes ___________ in the definitive host and ____________ in the intermediate host

A

The Sarcocystis neurona life cycle includes gametogony in the definitive host and schizogony in the intermediate host

13
Q

In the normal **Sarcocystis neurona life cycle, where would you find bradyzoites?

A

muscle tissue

14
Q

Where do schizonts develop in the accidental **Sarcocystis neurona life cycle?

A

Schizonts develop in the horse’s neurons, NOT in muscle cells

15
Q

Describe any pathogenesis in the final host for Sarcocystis neurona

A

non-pathogenic

16
Q

Describe any pathogenesis in the intermediate host for Sarcocystis neurona

A

pathogenic effect due to 2nd stage scchizogony in vascular endothelium

17
Q

Describe any pathogenesis in the accidental host for Sarcocystis neurona

A

pathogenic due to 3rd stage schizogony in nervous tissue rather than muscle

18
Q

Describe any clinical signs in horses as a result of Sarcocystis neurona

A
  • Progressively debilitating disease affecting the CNS, brain, brainstem, and spinal cord
    • Unusual or atypical lameness, or seizures
    • Difficulty with standing, walking, or swallowing
    • May progress very rapidly
19
Q

What is the most commonly diagnosed infectious equine neurologic disease in America?

A

Equine Protozoal Myeloencephalitis (EPM)

20
Q

How would you diagnose Sarcocystis neurona in a horse before death?

A

clinical signs, western blotting, PCR of cerebral spinal fluid

21
Q

Describe treatment & prevention options for Sarcocystis neurona

A

Opossum control is CRITICAL. Prevent opossums from defecating where horses are fed.

Anti-protozoals can rid horse of Sarcocystis neurona organisms, but CNS damage may remain

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