Ch 10: Advnced Skin Disorders and Skin in Distrress Flashcards Preview

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Flashcards in Ch 10: Advnced Skin Disorders and Skin in Distrress Deck (18)
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1
Q

Second-degree sunburn. May involve a blister where epidermolysis is occurring: the epidermis is lifting off the dermis. No esthetic service should be performed because of heightened risk of infection.

A

Stage 2 Skin Injury

2
Q

Third-degree burn, extends into the dermal tissue and classified as a subcutaneous injury.

A

Stage 3 Skin Injury

3
Q

The deepest injury, involves muscle and bone exposure.

A

State 4 Skin Injury

4
Q

Healing by primary intention; sutures, staples, butterfly stitches and superficial adhesive agents. Literally means to close the wound.

A

Approximating the wound

5
Q

Inflammatory Phase, Proliferative Phase, Maturation Phase

A

Stages of the Inflammatory Response

6
Q

When can you wear sunscreen for wound healing?

A

7-10 days after initial treatment

7
Q

What typically happens when a person suffers from “inhalation of plume”?

A

Respiratory irritation

8
Q

Name 3 things that long-term sun exposure can cause to the skin

A

Hyper-pigmentation, Chloasma (liver spots), tinea versi color (sun fungus, white patches)

9
Q

Long-term damage to the skin caused by uv light. Collagen and elastin fibrils in the dermis begin a process called cross-linking, or collapsing because of cumulative effects of cell damage from repeated sun exposure, causing the support system of the skin to also collapse= wrinkling, sagging, and elastosis.

A

Dermatoheliosis

10
Q

A rough area of sun-damaged skin, indicated by dysplastic cell growth (abnormal growth.) Found on face of individuals with chronic sun exposure and light skinned people. Red, or pigmented, rough patches, and can be crusty, scaly, and rough to the tough. Feels like needles and splinters. Temples, forehead, hairline, ears, and hands. Because they are dysplastic cells, they can become cancerous and are often referred to as precancerous. The treatment would be to curet (dig out) or cut off.

A

Actinic Keratosis and Treatment

11
Q

A asymmetric. Usually grows to one side of the lesion and is uneven
B border. Uneven and not smooth
C color. Dark brown or black and are usually splotchy and not of one color
D diameter. At east the size of a pencil eraser or larger. Regular moles are usually smaller than this
E evolving moles. Changes may include darkening or variations in color, moles that itch or hurt, and changes in shape or growth of the mole

A

ABCDEs of Skin Cancer

12
Q

Papule, pustule, and nodule

A

Examples of Inflammatory Acne Lesions

13
Q

What happens when a closed comedo grows large enough to tear the follicle wall?

A

Stimulates the immune system to investigate, releasing white blood cells into the area. These white blood cells arrive via the blood vessels causing the lesion to become red=inflammatory lesion

14
Q

What results when a hair is trying to force its way out of a follicle and irritation occurs?

A

Keratosis Polaris; red, pink, sand paper texture, white heads. Treat with alpha hydroxy lotions and gels, and a mild mechanical exfoliating cleanser. Appears on arms for adults and cheeks on children.

15
Q

Only to the epidermis, or superficial level. A mild sunburn is clarified as an epidermal injury.

A

Stage 1 Skin Injury

16
Q

The period immediately after injury. This is a period of vasoconstriction that lasts several minutes, which establishes hemostasis (control of bleeding), followed by a couple days of vasodilatation that allows a build up of necessary cells and protein to repair deeper wounds. Fibroblasts are stimulated to regulate the production of collagen.

A

Inflammatory Phase

17
Q

Usually begins about one week after injury or incision ( a lot going on to heal.) An increase in wounds vascularity to provide critical nutrients and oxygen to sustain the metabolism of the healing wound, particularly the deposits of collagen. Wound contracture begins a the tail end of this Phase.

A

Proliferative Phase

18
Q

Remolding, is the final phase of wound healing is characterized by an increase in strength w/o increase in collagen content. By three to six weeks, the maximum amount of collagen has been laid down in the wound, however this collagen is the n remolded and aligned to maximum wound tensil strength. Special enzymes ensure that the breakdown and production of collagen is balanced to keep a constant amount of collagen in the wound. Cross-linking= increased strength. One month=40% of strength, usually peaks at 80% of original strength.

A

Maturation Phase